HBOT hyperbaric chambers macy pan hyperbaric oxygen chamber sitting type portable chamber 1.3ata
Brief Introducing of Hyperbaric Oxygen Chamber
Brand MACY-PAN Oxygen Chamber is a multifunctional chamber for people to lie inside and enjoy a pleasant treatment which is short of HBOT (Hyperbaric Oxygen Therapy) at 1.3ATA high pressure. It can used in clinic, SPA, health center, gym, beauty and health salon, oxygen bar, diving spots, hotel and home. And it’s easy for one person to operate it, the using time is about 30-60 minutes.
What is Hyperbaric Oxygen Therapy (HBOT)?
Hyperbaric oxygen therapy (HBOT) is a medical treatment which enhances the body's natural healing process by inhalation of 100% oxygen in a total body chamber, where atmospheric pressure is increased and controlled. It is used for a wide variety of treatments usually as a part of an overall medical care plan.
Under normal circumstances, oxygen is transported throughout the body only by red blood cells. With HBOT, oxygen is dissolved into all of the body's fluids, the plasma, the central nervous system fluids, the lymph, and the bone and can be carried to areas where circulation is diminished or blocked. In this way, extra oxygen can reach all of the damaged tissues and the body can support its own healing process. The increased oxygen greatly enhances the ability of white blood cells to kill bacteria, reduces swelling and allows new blood vessels to grow more rapidly into the affected areas. It is a simple, non-invasive and painless treatment.
What Condition You Can't Use the Chamber for Treatment?
· If you have any cold or flu symptoms, fever, sinus or nasal congestion, or chest congestion.
· If there is a possibility that you may be pregnant.
· If there has been a change in any of your medications.
· If you have skipped a meal prior to your HBO treatment.
· If you are diabetic and did not take your insulin prior to your treatment.
· If you have any concerns or anxiety.
How Does Hyperbaric Oxygen Help Brain Injury or Stroke?
Hyperbaric oxygen therapy (HBOT) is a medical treatment which enhances the body’s natural healing process by inhalation of 100% oxygen in a total body chamber, where atmospheric pressure is increased and controlled. It is used for a wide variety of treatments usually as a part of an overall medical care plan.
When cells in the brain die, either from trauma or lack of oxygen, blood plasma leaks out into surrounding brain tissue causing swelling and reducing blood flow. These otherwise normal cells go dormant because they can't function without the appropriate amount of oxygen. HBOT dramatically increases the oxygen carried in the blood plasma, making oxygen available to heal damaged capillary walls, preventing plasma leakage and reducing swelling. As the swelling decreases, blood flow can be restored to the dormant tissue (neovascularization) and these cells then have the potential to function again.
Why Is Oxygen So Important?
Every day an average adult consumes 2 kg of food, 1 liter of water and almost 550 liters of oxygen. 90% of our daily energy comes from oxygen and the rest food and water. our brain is 2% of the total weight and uses of 25% of the oxygen we breath. We need this oxygen for the energy cycle that sustains life. When we do not have enough oxygen in our body tissues a series of events occur that if not corrected lead to disease conditions, either infection, tissue destruction or both. If there is low oxygen in tissues (hypoxia) there is a short window of opportunity to correct it. An excellent method to correct tissue hypoxia is by using a hyperbaric chamber.
What do you feel inside a hyperbaric chamber?
When the chamber is pressurization, your ears may feel pressure changes. You might feel a little painful in ears. To equalize the pressure and avoid the feeling of fullness in ears, you can yawning, swallowing or “blow the nose”. Other than this ear pressure there are no usual or different sensations.
How does being inside a pressurized chamber give us more oxygen?
When we are inside a chamber pressurized at 1.3 or 1.5 times of the normal air pressure it may not feel different, but breathing pure oxygen in such a chamber gives 7-8 times the regular amount of oxygen. Henry’s Law-Henry law states that for a gas-liquid interface the amount of gas that dissolves in the liquid is proportional to its partial pressure. The pressurized oxygen dissolves directly into all body fluids. In a few minutes this extra oxygen builds up tissue oxygen levels far above normal. This action has been scientifically proven to stimulate healing.
What improvements can I expect to see?
This is one of those questions which has so many different variables and answers we will have to generalize. We will answer this question from general experience and with the understanding that no 2 people get the same results. It is very rare that we do not use see improvements. What we are looking for is an improvement in the quality of life for anyone.
When will I know how many sessions of hbot are enough?
You will not. What you will be able to notice is whether you are continuing to get improvements. A clinical study on 1730 subjects shows the need of around 15 sessions for acute injuries and 40 for chronic ones. If you saw results during the first 4- sessions, do another 40. If improvement continues, do another 40. Etc. Etc. You are a better judge of the number of sessions needed than any Doctor.
What about Oxygen Toxicity?
Oxygen Toxicity is not a probelm at pressure below 1.5ATA. Many times stress is mistakes for oxygen toxicity. Occasionally, there may be a patient who is more sensitive to Oxygen than others and pressures may need to be adjusted to meet the needs of the patient.
*MEDICAL
*BEAUTY SALON&SPA
*GYM&TRACIING RECOVERY
*HOME USE&HEALTH CARE
Hyperbaric Oxygen Chamber can be used for everyone almost to get the benefits of oxygen.
POTENTIAL MECHANISMS OF HBOT
Researches on HBOT in experimental TBI studies have clarified diverse mechanisms leading to neuroprotection. Many of the pathways work parallel, or together, to induce neuroprotection in the brain. These mechanisms include: 1) increasing tissue oxygenation; 2) reducing inflammation; 3) decreasing apoptosis; 4) reducing ICP; 5) promoting neurogenesis and angiogenesis. For the purpose of this review, a brief summary of the recent discoveries in the mechanism of HBOT will be discussed. Table 1 lists most recent exciting discoveries in animal models, and Figure 1 summarizes the potential mechanisms involved in HBOT.
HBOT increases tissue oxygenation
Henry's law states that the amount of gas dissolved in a liquid or tissue is proportional to the partial pressure of that gas in contact with the liquid or tissue. This is the basis for increased tissue oxygen tensions with HBO treatment. In physiological condition, most oxygen carried in the blood is bound to hemoglobin, which is 97% saturated at atmospheric pressure. The other oxygen is carried in solution, and this portion is increased with the pressure due to Henry's Law. When breathing normobaric air, arterial oxygen tension is approximately 100 mmHg, and tissue oxygen tension approximately 55 mmHg. However, 100% oxygen at 3 ATA can increase arterial oxygen tensions to 2,000 mmHg, and tissue oxygen tensions to around 500 mmHg (Ratzenhofer-Komenda et al., 2006). The marked increased oxygen tension gradient from the blood to metabolizing cells is a key mechanism by which hyperoxygenation of arterial blood can improve effective cellular oxygenation even at low rates of tissue blood flow (Thom, 2011). And as the oxygen is in solution, it can reach physically obstructed areas where red blood cells cannot pass. HBO (1.5 ATA for 60 minutes) significantly increases brain tissue oxygen pressure (pO2) in both injured and sham-injured rats (Daugherty et al., 2004; Niklas et al., 2004). Results of several studies suggest that increasing brain tissue oxygenation contributed to reduced mortality and improved outcome of TBI patients (Rockswold et al., 2010, 2013).
HBOT suppresses inflammation
The acute inflammatory response plays an important role in secondary brain damage after TBI, which is characterized by cytokine release, neutrophil activation and microvascular adherence. Reducing inflammation is essential for the treatment of TBI. HBO has been shown to suppress inflammation in many studies (Vlodavsky et al., 2006; Lin et al., 2012; Zhang et al., 2014a; Meng et al., 2016a, b). When healthy humans are exposed to HBO at 2.8 to 3.0 ATA for at least 45 minutes, the ability of circulating neutrophils to adhere to target tissues is temporarily inhibited (Kalns et al., 2002). In a blast-induced traumatic brain injury model in rabbits, HBOT (2 ATA for 60 minutes) administrated at 12 hours after injury reduced the RNA and protein levels of caspase-3, interleukin-8 and tumor necrosis factor-α (Zhang et al., 2014a). In the early stage of TBI in rats, HBOT improved outcomes and reduced inflammation by increasing anti-inflammatory cytokine interleukin-10 (Lin et al., 2012; Chen et al., 2014), attenuating microgliosis and decreasing the level of tumor necrosis factor-α (Lim et al., 2013), and decreasing the expression of matrix metalloproteinase-9 (Vlodavsky et al., 2006). Recently, Meng et al. (2016a) showed HBOT significantly increased the expression of nuclear factor (erythroid-derived 2)-like 2 and heme oxygenase-1, and inhibited the expression of Toll-like receptor 4 and nuclear factor-kappaB in a rat TBI model (Meng et al., 2016b). The inhibitory effect of HBOT on inflammation closely associated with the decreased brain edema, blood-brain barrier leakage, cell apoptosis and improved neurological disorders after TBI.
HBOT decreases apoptosis
Apoptosis occurred in the brain tissues from the first few hours to weeks after TBI (Rink et al., 1995). The growth and progression of TBI lesions depend significantly on the developments in traumatic penumbra area and perilesional region, where neuronal apoptosis occurs. Inhibition of apoptosis becomes a therapeutic strategy to preserve brain tissues and promote functional recovery. In previous studies, HBOT reduced brain infarction and improved neurological deficits by preventing neuron apoptosis in ischemic stroke (Zhou et al., 2000; Yin et al., 2003; Li et al., 2005; Lou et al., 2006; Peng et al., 2009) and hypoxia-ischemia (Calvert et al., 2002, 2003; Liu et al., 2013) animal models. The neuroprotective, anti-apoptotic effects of HBOT in the development of secondary brain damage after TBI have been extensively investigated. Palzur et al. (2004, 2008) proved that HBOT reduced apoptosis in dynamic cortical deformation rats. HBOT suppressed the activation of the mitochondrial mediated apoptotic pathway by inducing the expression of Bcl-2 and preserving mitochondrial integrity (Palzur et al., 2008). The same mechanism was observed that HBOT increased expression of anti-apoptotic proteins (Bcl-2 and Bcl-xl) and decreased apoptosis in rat models of TBI (Vlodavsky et al., 2005; Liu et al., 2006; Xu et al., 2012). These results suggest that the neuroprotective effects of HBO are at least partially mediated by the reduction of apoptosis.
HBOT reduces ICP
Critical elevation of ICP represents the leading cause of morbidity and mortality in patients suffering from severe TBI (Horn et al., 1999). One of the important mechanisms of HBOT for severe brain injury is to lower ICP. Brown et al. (1998) first reported that HBO at 2 ATA for 60 minutes lowered ICP during the first 15 minutes of therapy in head-injured patients, however, rebound elevation were seen during or after therapy (Brown et al., 1988). A definite positive effect of HBOT on ICP was achieved by Rogatsky et al. (2005). HBOT (2.5 ATA for 60 minutes) applied within 24 hours ameliorated the outcome and reduced ICP by decreasing endothelin, improving the blood velocity of middle cerebral artery and decreasing cerebral vascular resistance in severe TBI patients (Ren et al., 2001b). Mechanisms of HBOT reducing ICP can also be related to the subsequent sustained improvement in tissue metabolism of the traumatized brain, as shown by Rockswold et al. (2001). The same conclusion was drawn when applied HBO to rats after severe fluid percussion brain injury. HBOT significantly diminished ICP elevation rate and decreased mortality when HBO was administrated within 2 hours after severe trauma (Rogatsky et al., 2005).
HBOT promotes neurogenesis and angiogenesis
Many studies have reported that multiple HBOT could improve neurological deficits and cognitive impairments at the acute stage (Lin et al., 2012) and at late chronic stages, months to years after TBI (Brown et al., 1988; Contreras et al., 1988; Horn et al., 1999; Ren et al., 2001b; Daugherty et al., 2004; Harch et al., 2007, 2009, 2012; Kernie and Parent, 2010; Brkic et al., 2012; Xu et al., 2012; Boussi-Gross et al., 2013). The therapeutic effects of long-term HBOT may be associated with multifaceted repair, including activation of angiogenesis and triggering of neuroplasticity, and induce proliferation and differentiation of neuronal stem cells. When HBO (2 ATA for 60 minutes twice a day for 3 consecutive days) was given within 3 hours after injury in fluid percussion model of TBI in rats, there was a significant increase in newborn endothelia cells, neurons and glial cells at 4 days after TBI (Lin et al., 2012). Ten exposures of HBO (2.5 ATA for 60 minutes for 10 days) can intensify neuroplastic responses by promoting axonal sprouting and synapse remodeling, which contributes to the recovery of locomotor performances in TBI rats (Brkic et al., 2012); 40-day series of 80 HBOTs (1.5 ATA for 90 minutes each time) caused an increase in contused hippocampus vascular density and an associated improvement in cognitive function (Harch et al., 2007). Activation of several signaling pathways and transcription factors have been suggested to play an important role in HBOT-induced neurogenesis, including Wnt, hypoxia-inducible factors and cAMP response element-binding (Mu et al., 2011).
Alertness.
| Improves metabolism and aids digestion.
| Reduces fatigue and improves sleeping patterns. |
MACY-PAN:
---- Professional Manufacturer of Portable Hyperbaric Chamber in China for 8 years.
-----Most affordable and Competitive Price on global market.
-----Accept Custom Made(OEM & ODM) for your requests.
-----Fast Delivery Time.
-----All the chambers have been carefully tested.
-----Warranty 1 year.
We accept EXW,FOB,CIF,DDU,DDP as Price Terms, DHL,FEDEX,TNT,UPS as air express.
Here is one copy of packing list.
| PACKING LIST | |||||
| Case No | N.W | G.W. | Art No | SET/PCS | Volume (M3) |
| 1 | 30kg | 31kg | TPU Inflatable Bag | 1 | 88x58x49cm=0.25CBM |
| 2 | 18Kg | 19Kg | Oil Free Air Compressor | 1 | 53x36x41cm=0.08CBM |
| 3 | 30Kg | 34Kg | Piston Compressor | 1 | 50x45x80cm=0.18CBM |
| 4 | 4Kg | 5Kg | Dehumidifier | 1 | 27x21x33cm=0.02CBM |
| TOTAL | 0.53CBM | ||||
| NW:82KG | |||||
| GW:89KG | |||||
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